Gp53

Researchers report that Gp53, a transcription factor from a bacteriophage, suppresses cell division and alters the physiology of Pseudomonas aeruginosa. The finding, published in Nature, sheds light on how phage proteins reprogram host bacteria, with potential implications for understanding phage-host interactions and for antimicrobial research targeting Pseudomonas, a common multidrug-resistant pathogen.